Cytochrome c release and apoptosis induced by mitochondrial targeting of nuclear orphan receptor TR3.

نویسندگان

  • H Li
  • S K Kolluri
  • J Gu
  • M I Dawson
  • X Cao
  • P D Hobbs
  • B Lin
  • G Chen
  • J Lu
  • F Lin
  • Z Xie
  • J A Fontana
  • J C Reed
  • X Zhang
چکیده

TR3, an immediate-early response gene and an orphan member of the steroid-thyroid hormone-retinoid receptor superfamily of transcription factors, regulates apoptosis through an unknown mechanism. In response to apoptotic stimuli, TR3 translocates from the nucleus to mitochondria to induce cytochrome c release and apoptosis. Mitochondrial targeting of TR3, but not its DNA binding and transactivation, is essential for its proapoptotic effect. Our results reveal a mechanism by which a nuclear transcription factor translocates to mitochondria to initiate apoptosis.

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عنوان ژورنال:
  • Science

دوره 289 5482  شماره 

صفحات  -

تاریخ انتشار 2000